Disease Model of GATA4 Mutation Reveals Transcription Factor Cooperativity in Human Cardiogenesis [ATAC-Seq]
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE85630
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资源简介：
Heterozygous mutations in GATA4 cause congenital heart defects and cardiomyopathy through unknown mechanisms. To gain insights into the open chromatin status during human cardiac development due to GATA4 heterozygosity, we performed ATAC-seq of wildtype and GATA4-G296S diseased cardiac progenitors. ATAC sequencing of wildtype and diseased pluripotent stem cell derived cardiomyocytes
创建时间：
2019-05-15
