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Identification of transcript dysregulation in rats with a neuropathic pain, treated or not with vitamin D

ChEBI2018-04-29 更新2026-07-12 收录

神经病理机制

药物干预转录组

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-6801 数据链接链接失效反馈

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资源简介：

Identification of transcript dysregulation in rats with a neuropathic pain, treated or not with vitamin D

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创建时间：

2018-04-29

神经病理机制

背根神经节基因表达

Chronic neuropathic pain is a major morbidity of neural injury, yet its mechanisms are incompletely understood. Hypersensitivity to previously non-noxious stimuli (allodynia) is a common symptom. Here

NIAID Data Ecosystem110

Additional file 5 of The interictal transcriptomic map of migraine without aura

偏头痛转录组学

神经病理机制

Supplementary Material 5

DataCite Commons2025-05-12 更新60

Table_1_Tandem Mass Tag-Based Quantitative Proteomic Analysis Reveals Pathways Involved in Brain Injury Induced by Chest Exposure to Shock Waves.DOCX

创伤蛋白质组学

神经病理机制

Recurrent chest blast exposure can lead to brain inflammation, oxidative stress, and mental disorders in soldiers. However, the mechanism that underlies brain injury caused indirectly by chest blasts

NIAID Data Ecosystem50

Effective control of neuropathic pain by interrupting C/EBPβ-CLEC7A cross-talk-mediated NLRP3 inflammasome-dependent pyroptosis. Effective control of neuropathic pain by interrupting C/EBPβ-CLEC7A cross-talk-mediated NLRP3 inflammasome-dependent pyroptosis

神经病理机制

脊髓与骨髓基因表达谱

Neuropathic pain (NP) is a complex chronic pain due to the nervous system damage or diseases.During NP development and progression, the neuroinflammation has been observed along the pain pathways from

NIAID Data Ecosystem90

Disease-associated astrocyte epigenetic memory promotes CNS pathology [For 3 times stimulation and Germ Free Mice]

胶质细胞表观遗传学

神经病理机制

Disease-associated astrocyte epigenetic memory promotes CNS pathology In this study we generated ATAC-Seq data from TNF-Alpha and PBS stimulated astrocytes from Germ Free Mice and Normal Mice. Normal

NIAID Data Ecosystem50

Identification of transcript dysregulation in rats with a neuropathic pain, treated or not with vitamin D

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