官方服务：
资源简介：
https://genome.cshlp.org/content/27/2/259.full
应用场景：
创建时间：
2019-01-07
相关数据集
Control ChIP-seq from activated B cell (ENCSR003FJP)
For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Pages/Research/ENCODE/ENCODE_Data_Use_Policy_for_External_Users_03-07-14.pdf https://www.e
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TF ChIP-seq from whole organism (ENCSR861RPB)
ChIP-seq on transgenic flies expressing CG13775-eGFP fusion proteins. The IP was performed using an anti-GFP antibody. For data usage terms and conditions, please refer to http://www.genome.gov/27528
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ZXDA/B and CENP-A/B Chromatin immunoprecipitation sequencing (ChIP-seq) in human colon carcinoma cells.. ZXDA/B and CENP-A/B Chromatin immunoprecipitation sequencing (ChIP-seq) in human colon carcinoma cells.
The goal of this experiment was to investigate the role of the FBXO38 ubiquitin ligase in the regulation of centromeric proteins deposition in HCT116 cell line.
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Control ChIP-seq from K562 (ENCSR956GFH)
K562 immortalized cell line stably expressing C-terminal eGFP-ZNF778 fusion protein. For data usage terms and conditions, please refer to http://www.genome.gov/27528022 and http://www.genome.gov/Page
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MLL3 Loss Drives Metastasis by Promoting a Hybrid Epithelial-Mesenchymal Transition State [ChIP-seq UTX and MLL4]. MLL3 Loss Drives Metastasis by Promoting a Hybrid Epithelial-Mesenchymal Transition State [ChIP-seq UTX and MLL4]
Phenotypic plasticity associated with the hybrid epithelial-mesenchymal transition (EMT) state is crucial to metastatic seeding and outgrowth. However, the mechanisms controlling the induction of hybr
NIAID Data Ecosystem10
