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Transcriptional re-programming of primary human macrophages by IRF-3 and IRF-7

ChEBI2008-11-29 更新2026-07-21 收录

巨噬细胞免疫调节

转录重编程

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-12120 数据链接链接失效反馈

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资源简介：

Transcriptional re-programming of primary human macrophages by IRF-3 and IRF-7

应用场景：

创建时间：

2008-11-29

巨噬细胞免疫调节

表观遗传时序分析

Deletion of Arntl disrupted temporal inflammatory response in macrophage. Mechanistically, the acetylation status of lysine 27 of histone 3 (H3K27ac) was enhanced at the PU.1-containing enhancers in A

NIAID Data Ecosystem60

Gene expression profiling of human MSC-educated macrophages vs. classical macrophages from bone marrow and blood. Homo sapiens

巨噬细胞免疫调节

组织修复机制

In comparison to MØs, MEMs have increased expression of the inhibitory molecules PD-L1, PD-L2, in addition to markers of alternatively activated macrophages: CD206 and CD163. RNA-Seq analysis of MEMs,

NIAID Data Ecosystem50

Transcriptional reprogramming of skeletal muscle stem cells by the niche environment

骨骼肌干细胞

转录重编程

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

NIAID Data Ecosystem30

SCL and LMO1 reprogram thymocytes into self-renewing cells.

白血病前干细胞机制

转录重编程

The SCL and LMO1 oncogenic transcription factors reprogram thymocytes into self-renewing pre-leukemic stem cells (pre-LSCs). Here we report that SCL directly interacts with LMO1 to activate the transc

NIAID Data Ecosystem60

Additional file 1 of Identification of sulfur metabolism-related gene signature in osteoarthritis and TM9SF2’s sustenance effect on M2 macrophages' phagocytic activity

关节炎分子机制

巨噬细胞免疫调节

Additional file 1. Table 1. Gene lists for analysis.

NIAID Data Ecosystem40