BTG1 regulates glucocorticoid receptor autoinduction in acute lymphoblastic leukemia
收藏官方服务：
资源简介：
BTG1 regulates glucocorticoid receptor autoinduction in acute lymphoblastic leukemia
应用场景：
创建时间：
2010-06-23
相关数据集
The molecular basis of T-PLL is an actionable perturbation of TCL1/ATM- and epigenetically instructed damage responses [murine SNP array]
T-cell prolymphocytic leukemia (T-PLL) is a rare and poor-prognostic mature T-cell malignancy. To address its incomplete molecular concept, we integrated large-scale profiling data of alterations in g
NIAID Data Ecosystem60
Circularized chromosome conformation capture (4C-seq) in A549 and U2OS cells stably expressing rat GRα. Circularized chromosome conformation capture (4C-seq) in A549 and U2OS cells stably expressing rat GRα
To investigate the interaction landscape at the GILZ locus we performed 4C-seq in A549 (ATCC CCL-185) and U2OS cells stably expressing rat GRα (Rogatsky et al. , Mol Cell Biol, 1997. 17(6): p. 3181-93
NIAID Data Ecosystem40
Table_1_LUMAN/CREB3 Plays a Dual Role in Stress Responses as a Cofactor of the Glucocorticoid Receptor and a Regulator of Secretion.docx
LUMAN/CREB3, originally identified through its interaction with a cell cycle regulator HCFC1, is a transcription factor involved in the unfolded protein response during endoplasmic reticulum stress. P
NIAID Data Ecosystem20
Acute leukemias of different lineages have similar MLL gene fusions encoding related chimeric proteins resulting from chromosomal translocation.
The MLL gene, on human chromosome 11q23, undergoes chromosomal translocation in acute leukemias, resulting in gene fusion with AF4 (chromosome 4) and ENL (chromosome 19). We report here translocation
PubMed Central1993-09-15 更新40
Myb overexpression synergizes with loss of Pten and is a dependency factor and therapeutic target in T-cell acute lymphoblastic leukemia
T-lineage acute lymphoblastic leukemia (T-ALL) is an aggressive hematological malignancy that accounts for 10%–15% of pediatric and 25% of adult ALL cases. Although the prognosis of T-ALL has improved
NIAID Data Ecosystem20
