TP63 attenuates EMT potential in an experimental prostate cell model

ChEBI2013-03-17 更新2026-07-21 收录

前列腺癌分子机制

上皮-间质转化调控

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-1471 数据链接链接失效反馈

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资源简介：

TP63 attenuates EMT potential in an experimental prostate cell model

创建时间：

2013-03-17

TFEB promotes prostate cancer progression via regulating ABCA2-dependent lysosomal biogenesis

前列腺癌分子机制

基因表达调控

The goals of this study is to find effect of overexpression-TFEB in prostate cancer cell (DU145 cell line). Overall design: To further explore the potential molecular mechanisms of how TFEN affects ca

NIAID Data Ecosystem80

Constitutively Active Androgen Receptor Variants Upregulate Expression of Mesenchymal Markers in Prostate Cancer Cells

前列腺癌分子机制

激素受体功能

Androgen receptor (AR) signaling pathway remains the foremost target of novel therapeutics for castration-resistant prostate cancer (CRPC). However, the expression of constitutively active AR variants

NIAID Data Ecosystem60

Co-expression of genes with ERG in prostate cancer cell lines. Homo sapiens

前列腺癌分子机制

基因共表达网络

ERG overexpression is the most frequent molecular alteration in prostate cancer. We analyzed different stages of prostate cancer to identify genes that were coexpressed with ERG overexpression. In pri

NIAID Data Ecosystem20

TACC1.RARg.AR.veh.bw

前列腺癌分子机制

CUT&Tag表观遗传学分析

CUT&RUN for AR in 22Rv1 cells stably expressing RARgamma and TACC1 and treated with vehicle

Figshare2024-02-19 更新80

ERG-mediated coregulator complex formation maintains androgen receptor signaling in prostate cancer [PolII ChIP]

前列腺癌分子机制

雄激素受体信号

We report the effects of ERG on prostate tumorigenesis, ERG-mediated oncogene addiction, and downstream AR signaling pathways. We determined that ERG facilitates AR-signaling and mediates transformati

NIAID Data Ecosystem30

TP63 attenuates EMT potential in an experimental prostate cell model

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