Type 3 inflammation drives liver fibrosis by enhancing TGF-beta signaling through activation of MAPKS
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https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE119047
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资源简介：
Type 3 inflammation characterized by increased production of IL-22 is a common driver of liver fibrosis during chronic liver injury through enhancement of TGF-β signaling in a p38 MAPK dependent manner. Three lots of human primary hepatitic stellate cells. Each lot was stimulated for 48h with either PBS, TGFlo (0.1ng/mL), TGFhi (2,5ng/mL), IL-17A (40ng/mL), IL-17A + TGFlo, IL-22 (40ng/mL) or IL-22 + TGFlo. Each lot comes from a single donor.
创建时间：
2019-03-27
