Titin truncating variantsin hiPSC-cardiomyocytes inducepathogenic proteinopathy, sarcomeredefect and contractile dysfunction independent of the core contractilemachinery. Titin truncating variantsin hiPSC-cardiomyocytes inducepathogenic proteinopathy, sarcomeredefect and contractile dysfunction independent of the core contractilemachinery
收藏NIAID Data Ecosystem2026-03-12 收录
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This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
创建时间：
2021-09-01
