Genomewide profiling of H3K27ac following HDAC1/2 inhibition with romidepsin and evaluation of co-regulated mechanisms by HDAC2 and SMAD3 in BTSC genome.
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https://www.ncbi.nlm.nih.gov/sra/SRP401318
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To investigate the impact on global H3K27ac patterns following HDAC1/2 inhibition with romidepsin, we performed using ChIP-seq using formaldehyde crosslinked BT67 romidepsin treated versus vehicle treated cells. Overall design: To explore the cooperative mechanisms regulated by HDAC2 and SMAD3 in BTSC, we performed ChIP-seq using dual crosslinked BT67 cells.
创建时间：
2023-09-15
