Atypical inflammatory kinase IKBKE phosphorylates and inactivates FoxA1 to promote hepatic tumorigenesis
收藏NIAID Data Ecosystem2026-05-01 收录
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1062288
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资源简介：
our findings not only reveal FoxA1 as a bona fide substrate and negative nuclear effector of IKBKE in HCC progression, but also highlight a strategy to combat HCC by antagonizing IKBKE.
创建时间：
2024-01-08
