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The m6A reader YTHDF1 attenuates fulminant hepatitis via MFG-E8 translation in an m6A dependent manner

NIAID Data Ecosystem2026-05-01 收录

m6A修饰机制

急性重型肝炎分子病理

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE228282 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2023-08-21

RNA表观遗传学

m6A修饰机制

N6-methyladenosine (m6A) is the major type of RNA modi?cation that regulates RNA stability and gene expression, characterized by a conserved motif including 5'-(m6A)C-3'. However, the functional signi

NIAID Data Ecosystem110

The yeast RNA methylation complex consists of conserved yet reconfigured components with m6A-dependent and independent roles

表观转录组学

m6A修饰机制

N6-methyladenosine (m6A), the most abundant mRNA modification, is deposited in mammals/drosophila/plants by m6A methyltransferase complexes (MTC) comprising a catalytic subunit and at least five addit

NIAID Data Ecosystem70

Additional file 2 of Diagnostic value and immune infiltration characterization of YTHDF2 as a critical m6A regulator in osteoarthritic synovitis

骨关节炎滑膜炎免疫浸润

m6A修饰机制

Additional file 2: The list of macrophage-related genes.

Figshare2024-02-13 更新40

miCLIP in METTL14 R298P mutation knock-in cell clones

RNA表观遗传学

m6A修饰机制

N6-methyladenosine (m6A) is the major type of RNA modi'cation that regulates RNA stability and gene expression, characterized by a conserved motif including 5'-(m6A)C-3'. However, the functional signi

NIAID Data Ecosystem80

METTL3 suppresses pancreatic ductal adenocarcinoma progression through activating endogenous dsRNA-induced anti-tumor immunity

肿瘤免疫治疗

m6A修饰机制

Although immunotherapy improves clinical outcomes in several types of malignancies, as an immunologically cold tumor, pancreatic ductal adenocarcinoma is arrantly resistant to immunotherapy. However,

NIAID Data Ecosystem50

The m6A reader YTHDF1 attenuates fulminant hepatitis via MFG-E8 translation in an m6A dependent manner

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