首页/数据集/Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury.

Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury.

ChEBI2014-01-01 更新2026-07-21 收录

皮肤干细胞调控

表皮再生机制

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-50673 数据链接链接失效反馈

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资源简介：

Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury.

应用场景：

创建时间：

2014-01-01

皮肤干细胞调控

癌细胞机制

It is known that Rho GTPases control different aspects of the biology of skin stem cells (SSCs). However, little information is available on the role of its upstream regulators under normal and tumori

NIAID Data Ecosystem70

Lhx2 ChIP-chip from primary mouse keratinocytes

皮肤干细胞调控

伤口修复分子机制

The Lhx2 transcription factor plays essential roles in morphogenesis and patterning of ectodermal derivatives, as well as in controlling stem cell activity. Lhx2 is expressed in the hair follicle (HF)

NIAID Data Ecosystem50

Lhx2 ChIP-chip from primary mouse keratinocytes. Mus musculus

皮肤干细胞调控

毛囊周期

NIAID Data Ecosystem30

Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury [10wk skin]

皮肤干细胞调控

表皮再生机制

Transcriptional coactivator Mediator complex facilitates transcription of various transcription factors. Previously, we have generated Med1 conditional null mice, where a critical subunit of Mediator,

NIAID Data Ecosystem50

IRF2 is a master regulator of human keratinocyte stem cell fate [RNA-seq]. IRF2 is a master regulator of human keratinocyte stem cell fate [RNA-seq]

皮肤干细胞调控

转录因子功能研究

Resident adult epithelial stem cells maintain tissue homeostasis by balancing self-renewal and provision of differentiated cells. Human epidermal keratinocytes retain stem cell potential in vitro but

NIAID Data Ecosystem30

Ablation of coactivator Med1 regulates bulge keratinocyte stem cells and accelerates epidermal regeneration after injury.

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