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RNA-seq of brain tissues of WT and abcb4 knockout zebrafish at 2 months and 30 months

ChEBI2023-07-14 更新2026-07-12 收录

神经退行性机制

斑马鱼衰老转录组

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-12901 数据链接链接失效反馈

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资源简介：

RNA-seq of brain tissues of WT and abcb4 knockout zebrafish at 2 months and 30 months

应用场景：

创建时间：

2023-07-14

神经退行性机制

细胞应激通路

Ribosome stalling during translation has recently been shown to cause neurodegeneration, yet the signaling pathways triggered by stalled elongation complexes are unknown. To investigate these pathw

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AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons. AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons

神经退行性机制

功能障碍研究

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

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Additional file 15 of Comparative analysis of transcriptome remodeling in plaque-associated and plaque-distant microglia during amyloid-β pathology progression in mice

神经退行性机制

胶质细胞转录组学

Additional file 15: Table S3. Expression values for all expressed genes (in pseudocounts), related to Fig. 2.

Figshare2022-09-26 更新20

Characterization of Inducible Models of Tay-Sachs and Related Disease

GM2神经节苷脂沉积症模型

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Tay-Sachs and Sandhoff diseases are lethal inborn errors of acid β-N-acetylhexosaminidase activity, characterized by lysosomal storage of GM2 ganglioside and related glycoconjugates in the nervous sys

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Loss of TREM2 reduces hyperactivation of progranulin deficient microglia but not lysosomal pathology [14 months]

神经退行性机制

胶质细胞功能研究

GRN haploinsufficiency causes frontotemporal lobar degeneration and results in microglial hyperactivation, lysosomal dysfunction and TDP-43 deposition. To understand the contribution of microglial hyp

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RNA-seq of brain tissues of WT and abcb4 knockout zebrafish at 2 months and 30 months

资源简介：

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