N1EE Vesicles Promote Liver Metastasis by Inducing EMT and Activating Stellate Cells

Name: N1EE Vesicles Promote Liver Metastasis by Inducing EMT and Activating Stellate Cells
Creator: Harbin Medical University
Published: 2025-10-22T08:10:27+08:00

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肿瘤转移机制

肿瘤微环境

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资源简介：

N1EE Vesicles Promote Liver Metastasis by Inducing EMT and Activating Stellate Cells

提供机构：

Harbin Medical University

创建时间：

2025-10-22

Table_1_Comprehensive Genomic Characterization of Tumor Microenvironment and Relevant Signature in Clear Cell Renal Cell Carcinoma.xlsx

肿瘤微环境

基因组学

PurposeTo systematically investigate the characterization of tumor microenvironment (TME) in clear cell renal cell carcinoma (ccRCC), we performed a comprehensive analysis incorporating genomic altera

NIAID Data Ecosystem30

Supplemental Table S2. Primers from Activation of Wnt/β-Catenin in Ewing Sarcoma Cells Antagonizes EWS/ETS Function and Promotes Phenotypic Transition to More Metastatic Cell States

肿瘤转移机制

基因表达分析

Supplemental table S2. qRT-PCR primer sequences.

NIAID Data Ecosystem00

Development of a Folding Paper System To Enable the Analysis of Gene Profile of Short- and Long-Distance Cancer Cell Migration

肿瘤微环境

癌细胞迁移基因表达分析

In cancer metastasis, where mortality rates remain high despite advancements in medical treatments, understanding the molecular pathways and cellular dynamics underlying tumor spread is critical for d

NIAID Data Ecosystem00

Table_1_Risk Signature of Cancer-Associated Fibroblast–Secreted Cytokines Associates With Clinical Outcomes of Breast Cancer.xlsx

癌症预后预测

肿瘤微环境

Cancer-associated fibroblasts (CAFs) are key components in tumor microenvironment (TME). The secreted products of CAFs play important roles in regulating tumor cells and further impacting clinical pro

NIAID Data Ecosystem30

DataSheet1_Poly I:C-priming of adipose-derived mesenchymal stromal cells promotes a pro-tumorigenic phenotype in an immunocompetent mouse model of prostate cancer.zip

肿瘤微环境

间充质基质细胞

Introduction: Mesenchymal stromal cells (MSC) are envisioned as a potential cellular vehicle for targeted cancer therapies due to their tumor tropism and immune permissiveness. An obstacle in their us

NIAID Data Ecosystem00

N1EE Vesicles Promote Liver Metastasis by Inducing EMT and Activating Stellate Cells

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