首页/数据集/Rapidly induced fucosylation of intestinal epithelium sustains host-commensal symbiosis during sickness.

Rapidly induced fucosylation of intestinal epithelium sustains host-commensal symbiosis during sickness.

ChEBI2014-10-24 更新2026-07-21 收录

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宿主-微生物相互作用

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-60874 数据链接链接失效反馈

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资源简介：

Rapidly induced fucosylation of intestinal epithelium sustains host-commensal symbiosis during sickness.

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创建时间：

2014-10-24

宿主-微生物相互作用

遗传重组率

Wolbachia pipientis is an intracellular alphaproteobacterium that infects 40-60% of insect species and is well known for host reproductive manipulations. Although Wolbachia are primarily maternally tr

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Additional file 4 of Strain-level profiling with picodroplet microfluidic cultivation reveals host-specific adaption of honeybee gut symbionts

微生物菌株

宿主-微生物相互作用

Additional file 4: Dataset S1. Information of metagenome-assembled genomes.

Figshare2024-02-16 更新20

DataSheet_1_Metabolic reprogramming involves in transition of activated/resting CD4+ memory T cells and prognosis of gastric cancer.docx

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胃癌预后模型

BackgroundLittle is known on how metabolic reprogramming potentially prompts transition of activated and resting CD4+ memory T cells infiltration in tumor microenvironment of gastric cancer (GC). The

NIAID Data Ecosystem00

DataSheet_1_Low-density lipoprotein balances T cell metabolism and enhances response to anti-PD-1 blockade in a HCT116 spheroid model.zip

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PD-1联合治疗

IntroductionThe discovery of immune checkpoints and the development of their specific inhibitors was acclaimed as a major breakthrough in cancer therapy. However, only a limited patient cohort shows s

NIAID Data Ecosystem30

Genes in yeast two-hybrid screening.

宿主-病原体相互作用

免疫代谢

Intracellular bacterial pathogens exhibit heterogeneous replication rates within host macrophages, but the mechanisms by which they manipulate host factors for survival remain incompletely understood.

NIAID Data Ecosystem10

Rapidly induced fucosylation of intestinal epithelium sustains host-commensal symbiosis during sickness.

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