anti-IgLON5 disease characterisation

NIAID Data Ecosystem2026-05-02 收录

系统性自身免疫疾病

细胞死亡机制

数据链接：

https://www.omicsdi.org/dataset/pride/PXD052617 数据链接链接失效反馈

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资源简介：

Cytoskeletal disruption-induced calcium deregulation drives cell death in anti-IgLON5 disease

创建时间：

2025-09-05

Identification of Macrophage Inhibitory Factor as the Parthanatos Dependent AIF Associate Nuclease

细胞死亡机制

缺血性脑卒中分子靶点

Inhibition or genetic deletion of poly(ADP-ribose) polymerase-1 (PARP-1) is profoundly protective against a number of toxic insults in many organ systems1,2. In the brain the molecular mechanisms unde

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Caspase 3; the chelator treated groups o61–65 and o66–70 contains significantly more apoptotic neurons than the remaining groups.

凋亡研究

细胞死亡机制

Caspase 3; the chelator treated groups o61–65 and o66–70 contains significantly more apoptotic neurons than the remaining groups.

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None - E-cadherin endocytosis promotes non-canonical EGFR:STAT signalling to induce cell death and inhibit heterochromatinisation

细胞信号转导

细胞死亡机制

None - E-cadherin endocytosis promotes non-canonical EGFR:STAT signalling to induce cell death and inhibit heterochromatinisation

Figshare2025-07-21 更新30

Functional enrichment analysis results.

缺血再灌注损伤

细胞死亡机制

BackgroundIntestinal ischemia–reperfusion (II/R) injury is a severe clinical condition in which regulated cell death programs—including pyroptosis and necroptosis—have emerged as key drivers of tissue

Figshare2025-12-16 更新60

Strains used in this work.

细胞死亡机制

抗真菌剂作用机制

Thymidine starvation causes rapid cell death. This enigmatic process known as thymineless death (TLD) is the underlying killing mechanism of diverse antimicrobial and antineoplastic drugs. Despite dec

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anti-IgLON5 disease characterisation

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