Additional file 5 of NANOG controls testicular germ cell tumour stemness through regulation of MIR9-2

NIAID Data Ecosystem2026-05-01 收录

数据链接：

https://figshare.com/articles/dataset/Additional_file_5_of_NANOG_controls_testicular_germ_cell_tumour_stemness_through_regulation_of_MIR9-2/26714737 数据链接链接失效反馈

官方服务：

资源简介：

Additional file 5: Table S5. Predicted MIR9-2-5p targets

创建时间：

2024-05-01

Noncanonical mRNA decay by the endoplasmic-reticulum stress sensor IRE1-alpha promotes cancer cell survival [GRO-seq]. Noncanonical mRNA decay by the endoplasmic-reticulum stress sensor IRE1-alpha promotes cancer cell survival [GRO-seq]

内质网应激

癌细胞机制

Eukaryotic IRE1 mitigates endoplasmic-reticulum (ER) stress by orchestrating the unfolded-protein response (UPR). IRE1 spans the ER membrane, and signals through a cytosolic kinase-endoribonuclease mo

NIAID Data Ecosystem100

CD95L mRNA is toxic to cells [SmallRNA_CD95L.50hrs.Drosha_ko]

RNA干扰治疗

癌细胞机制

>80% of a large number of tested siRNAs and shRNAs targeting CD95 or CD95 ligand (CD95L) induce a form of cell death that is characterized by the simultaneous activation of multiple death pathways and

NIAID Data Ecosystem60

Data_Sheet_2_A Covalent Calmodulin Inhibitor as a Tool to Study Cellular Mechanisms of K-Ras-Driven Stemness.PDF

癌细胞机制

共价抑制剂开发

Recently, the highly mutated oncoprotein K-Ras4B (hereafter K-Ras) was shown to drive cancer cell stemness in conjunction with calmodulin (CaM). We previously showed that the covalent CaM inhibitor op

NIAID Data Ecosystem60

Velcrin-induced selective cleavage of tRNALeu(TAA) by SLFN12 causes cancer cell death [RNA-Seq]. Velcrin-induced selective cleavage of tRNALeu(TAA) by SLFN12 causes cancer cell death [RNA-Seq]

癌细胞机制

tRNA选择性切割

Velcrin compounds kill cancer cells expressing high levels of phosphodiesterase 3A (PDE3A) and Schlafen family member 12 (SLFN12) by inducing complex formation between these two proteins, but the mech

NIAID Data Ecosystem90

Type I IFNs promote cancer cell stemness by triggering the epigenetic regulator KDM1B. Type I IFNs promote cancer cell stemness by triggering the epigenetic regulator KDM1B

癌细胞机制

表观遗传调控

Cancer stem cells (CSCs) are immature, immortal cells within tumors, adept at resisting therapeutic pressure and responsible for local and distant disease recurrence. Non-genetic mechanisms of acquire

NIAID Data Ecosystem60

Additional file 5 of NANOG controls testicular germ cell tumour stemness through regulation of MIR9-2

资源简介：

相关数据集