KAP1 is a Chromatin Reader that Couples Steps of RNA Polymerase II Transcription to Sustain Oncogenic Programs

NIAID Data Ecosystem2026-03-11 收录

癌症表观遗传学

转录调控机制

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Collection of western blots and SDS-PAGE images

创建时间：

2020-02-20

ToxR bacterial transcriptional regulator bound to 25 bp toxT promoter DNA

转录调控机制

DNA相互作用

ToxR bacterial transcriptional regulator bound to 25 bp toxT promoter DNA Descriptor: Cholera toxin transcriptional activator, DNA (25-MER) Authors: Canals, A, Pieretti, S, Muriel, M, El Yaman, N, Fab

Protein Data Bank Japan2024-06-19 更新50

Dual regulation of open-complex formation and promoter clearance by Arc explains a novel repressor to activator switch.

转录调控机制

基因表达

In studies of variants of the P(ant) promoter of bacteriophage P22, the Arc protein was found not only to slow the rate at which RNA polymerase forms open complexes but also to accelerate the rate at

PubMed Central1996-08-20 更新30

A cohesin traffic pattern genetically linked to gene regulation [ChIP-Seq]

cohesin介导的染色质环挤压

转录调控机制

Cohesin-mediated loop extrusion folds interphase chromosomes at the ten to hundreds kilobases scale. This process produces structural features such as loops and topologically associating domains. We i

NIAID Data Ecosystem10

Nucleoporin 153 links nuclear pore complex to chromatin architecture by mediating CTCF and cohesin binding [RNA-Seq]

染色质三维构象调控

转录调控机制

Establishment of cell lineage specification, maintenance of cellular states and cellular responses to developmental cues rely on gene regulation and spatial genome organization during early developmen

NIAID Data Ecosystem50

Transcriptional regulation of aldo-keto reductase 1C1 in human colon cancer cells resistant to methotrexate. Homo sapiens

甲氨蝶呤耐药性

转录调控机制

The abstract of the associated publication (Selga E, Noé V, Ciudad CJ. Biochemical Pharmacology, 2008) is the following: While studying differentially expressed genes between sensitive and 10-5 M Meth

NIAID Data Ecosystem20

KAP1 is a Chromatin Reader that Couples Steps of RNA Polymerase II Transcription to Sustain Oncogenic Programs

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