Epigenomic analyses of clinical glioblastoma specimens

NIAID Data Ecosystem2026-05-01 收录

胶质母细胞瘤基因组学

脑肿瘤分子机制

数据链接：

https://www.omicsdi.org/dataset/biostudies-other/S-BSST1166 数据链接链接失效反馈

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资源简介：

Epigenomic GBM profiling to better understand early and late determinants of these tumors.

创建时间：

2023-08-08

Additional file 1: Table S1. of Evidence for L1-associated DNA rearrangements and negligible L1 retrotransposition in glioblastoma multiforme

胶质母细胞瘤基因组学

逆转录转座子

RC-seq output summary. Table S2. Polymorphic and somatic L1 mutations called by TEBreak. Table S3. List of somatic L1 mutations and PCR validation information. Table S4. GBM cell line transfection eff

NIAID Data Ecosystem50

Integrative Genome-wide Analysis of Glioblastoma.

胶质母细胞瘤基因组学

多组学数据整合

Glioblastoma multiforme shows multiple chromosomal aberrations, the impact of which on gene expression remains unclear. To investigate this relationship and to identify putative initiating genomic eve

NIAID Data Ecosystem40

Identification of SAT1 regulated genes in GBM cells by microarray

胶质母细胞瘤基因组学

微阵列转录组学

We aimed to identify SAT1 regulated genes in U87MG cell by knockdown of SAT1 with two different shRNAs, and then compared knockdown cells to control cells with shGFP U87 cells were infected with shRNA

NIAID Data Ecosystem30

Additional file 8 of Whole-genome sequencing of glioblastoma reveals enrichment of non-coding constraint mutations in known and novel genes

胶质母细胞瘤基因组学

非编码突变注释

Additional file 8: Table S7. Annotation of the NCCMs associated with the DYNC1I1 gene based on data from the ENCODE portal and the UCSC Browser databases.

Figshare2020-07-31 更新40

Prevalence of Copy-number neutral LOH in glioblastomas revealed by analysis of laser-microdissected tissues

胶质母细胞瘤基因组学

癌症遗传变异

Prevalence of Copy-number neutral LOH in glioblastomas revealed by analysis of laser-microdissected tissues

ChEBI2009-03-01 更新30