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To understand the transcriptome change in astrocytes after parental isolation
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Table_5_Common Dysregulation of Innate Immunity Pathways in Human Primary Astrocytes Infected With Chikungunya, Mayaro, Oropouche, and Zika Viruses.xlsx
Arboviruses pose a major threat throughout the world and represent a great burden in tropical countries of South America. Although generally associated with moderate febrile illness, in more severe ca
NIAID Data Ecosystem690
Genome-wide characterization of CREB-dependent transcriptional programs in astrocytes
Transcriptome profiling of astrocyte cultures treated with forskolin (FSK), noradrenaline (NE) or infected with adenovirus carrying a constitutively active form of CREB (VP16-CREB) vs. untreated or in
NIAID Data Ecosystem60
Next Generation Sequencing Facilitates Quantitative Analysis of primary cultured astrocytes treated with SB431542, Ro5-3335 and HLM006474 inhibitors.. Next Generation Sequencing Facilitates Quantitative Analysis of primary cultured astrocytes treated with SB431542, Ro5-3335 and HLM006474 inhibitors.
Purpose: The goals of this study are to investigate differential genes expression with RNA-seq in primary cultured astrocytes treated with the Tgf-beta1 receptor inhibitor SB431542, the Runx1 inhibito
NIAID Data Ecosystem40
Loss of TDP-43 in astrocytes leads to motor deficits by triggering A1-like reactive phenotype and tri-glial dysfunctions
The majority of patients with amyotrophic lateral sclerosis (ALS) have abnormal TDP-43 aggregates in the nucleus and/or cytosol of their surviving neurons and glia. Although accumulating evidence indi
NIAID Data Ecosystem80
T cell-specific deletion of TRAIL-receptor reveals a critical role for pathologic T cell activation and inflammation in experimental autoimmune encephalomyelitis
We isolated CD4 T cells from EAE mice with or without TRAIL treatment and analyzed by RNA-seq. We also activated naïve CD4 T cells by anti-CD3/CD28 antibodies in the presence or absence of TRAIL and s
DataMed40
