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Central role of SUMOylation in the regulation of chromatin interactions and transcriptional outputs of androgen receptor in prostate cancer cells

NIAID Data Ecosystem2026-05-02 收录

前列腺癌分子机制

染色质相互作用

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE211164 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2024-08-07

前列腺癌分子机制

基因表达调控

The goals of this study is to find effect of overexpression-TFEB in prostate cancer cell (DU145 cell line). Overall design: To further explore the potential molecular mechanisms of how TFEN affects ca

NIAID Data Ecosystem80

Constitutively Active Androgen Receptor Variants Upregulate Expression of Mesenchymal Markers in Prostate Cancer Cells

前列腺癌分子机制

激素受体功能

Androgen receptor (AR) signaling pathway remains the foremost target of novel therapeutics for castration-resistant prostate cancer (CRPC). However, the expression of constitutively active AR variants

NIAID Data Ecosystem60

Sarcosine Up-Regulates Expression of Genes Involved in Cell Cycle Progression of Metastatic Models of Prostate Cancer

前列腺癌分子机制

细胞周期调控

The effects of sarcosine on the processes driving prostate cancer (PCa) development remain still unclear. Herein, we show that a supplementation of metastatic PCa cells (androgen independent PC-3 and

Figshare2016-11-09 更新20

Additional file 7 of Human transcriptional interactome of chromatin contribute to gene co-expression

基因共表达

染色质相互作用

Additional file 7:Co-expressed gene pairs have more Hi-C interactions than all pairs. T tests are used to test the difference between Hi-C interactions of co-expressed gene pairs and all pairs. In co-

DataCite Commons2024-02-16 更新70

Paxillin regulates genomic networks in prostate cancer [PC3]

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基因调控网络

We report that the adaptor protein, paxillin, regulates some proliferative and apoptotic genes in the castration resistant prostate cancer cell line, PC3. PC3 cells were transfected with either Non-sp

NIAID Data Ecosystem50

Central role of SUMOylation in the regulation of chromatin interactions and transcriptional outputs of androgen receptor in prostate cancer cells

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