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TLE3 and TLE4-coordinated colonic macrophage-CD4+ T cell crosstalk maintains intestinal immune homeostasis

NIAID Data Ecosystem2026-05-01 收录

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免疫细胞互作网络

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE183236 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2023-04-10

基因表达

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The molecular mechanisms involved in host pathogen interactions at mucosal surfaces needs to be better understood in order to develop immune-mediated methods of protection against pathogens. Cholera t

NIAID Data Ecosystem50

A Preponderance of CCR5(+) CXCR4(+) Mononuclear Cells Enhances Gastrointestinal Mucosal Susceptibility to Human Immunodeficiency Virus Type 1 Infection

HIV与FIV感染机制

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The gastrointestinal mucosa harbors the majority of the body's CD4(+) cells and appears to be uniquely susceptible to human immunodeficiency virus type 1 (HIV-1) infection. We undertook this study to

PubMed Central20

Table_1_DC Respond to Cognate T Cell Interaction in the Antigen-Challenged Lymph Node.pdf

免疫细胞互作网络

T细胞极化机制

Dendritic cells (DC) are unrivaled in their potential to prime naive T cells by presenting antigen and providing costimulation. DC are furthermore believed to decode antigen context by virtue of patte

NIAID Data Ecosystem10

Bulk RNA-seq of murine ?dT17 and ?dT1 cells from the lamina propria and peripheral lymph nodes of wild-type mice

免疫细胞转录组学

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This experiment was designed to explore the transcriptional programs of ?dT17 and ?dT1 cells isolated from the lamina propria and peripheral lymph nodes. For this purpose, we sorted intestinal ?dT17 (

NIAID Data Ecosystem30

Table 4_Machine learning-driven dissection of the obesity-ccRCC interface: FCGR2A emerges as a central coordinator of tumor-immune crosstalk.xlsx

免疫细胞互作网络

肥胖相关癌症

IntroductionObesity is a well-established risk modifier for clear cell renal cell carcinoma (ccRCC), yet the molecular mechanisms linking these conditions remain incompletely characterized. MethodsW

NIAID Data Ecosystem10

TLE3 and TLE4-coordinated colonic macrophage-CD4+ T cell crosstalk maintains intestinal immune homeostasis

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