Role and function of STAT5 in BCR-ABL1 driven pre-B cells
收藏官方服务:
资源简介:
Role and function of STAT5 in BCR-ABL1 driven pre-B cells
应用场景:
创建时间:
2011-08-23
相关数据集
Time resolved quantitative phospho-tyrosine analysis reveals Bruton’s Tyrosine kinase mediated signaling downstream of the mutated granulocyte-colony stimulating factor receptors
Granulocyte-colony stimulating factor receptor (G-CSFR) controls myeloid progenitor proliferation and differentiation to neutrophils. Mutations in CSF3R (encoding G-CSFR) have been reported in patient
NIAID Data Ecosystem60
Gene expression data of BCR-ABL1 transformed B cell precursors from Dusp6 wild-type and Dusp6 knockout mice
To elucidate the mechanism of Dusp6-mediated pre-B cell survival signaling, we investigated the gene expression pattern in BCR-ABL1-transformed Dusp6+/+ and Dusp6-/- B cell precursors. BCR-ABL1 transf
NIAID Data Ecosystem20
Mutant forms of growth factor-binding protein-2 reverse BCR-ABL-induced transformation.
Growth factor-binding protein 2 (Grb2) is an adaptor protein that links tyrosine kinases to Ras. BCR-ABL is a tyrosine kinase oncoprotein that is implicated in the pathogenesis of Philadelphia chromos
PubMed Central1995-11-21 更新10
Transcriptome-wide targets of the RNA-binding protein MSI2 and its role in IL-6 signaling
The RNA-binding protein Musashi 2 (MSI2) is emerging to be an important regulator in cancer initiation, progression and drug resistance. Translocations and deregulation of this protein are diagnostic
NIAID Data Ecosystem40
Figure S1 - Targeting the Acute Promyelocytic Leukemia-Associated Fusion Proteins PML/RARα and PLZF/RARα with Interfering Peptides
Co-expression of PCC/POZ-GFP reverses IL3-independent growth in PCC/ABL- or POZ/ABL-positive BA/F3 cells. A–D Infected BA/F3 cells were cultivated for 6 days without IL3. The total cell count and FACS
Figshare2015-12-02 更新20



