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Diet-induced developmental acceleration independent of TOR and insulin in C. elegans.

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线虫发育调控

营养感知与发育

数据链接：

https://www.wormbase.org/resources/analysis/Microarray_Study.WBPaper00042258.ce.mr 数据链接链接失效反馈

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资源简介：

Diet-induced developmental acceleration independent of TOR and insulin in C. elegans.

应用场景：

转录因子ChIP-seq

线虫发育调控

We identified target genes for NHR-25 by ChIP-seq at L1 stage of C. elegans. Transcription factor genes were tagged with GFP and their expression examined at L1 stage. Since there are no direct target

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Neuronal IL-17 controls C. elegans developmental diapause through p53/CEP-1

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p53信号通路

Metazoan growth and development requires the coordination of cell cycle progression and metabolism with nutrient availability1-3. Here, we show that in C. elegans, amphid neurons regulate the animals’

NIAID Data Ecosystem20

Ethosuximide inhibited dauer formation in a high-density, starved population at 25°C.

线虫发育调控

药物表型筛选

***p<0.0001, comparisons are to WT with no drug treatment. p values determined by Student T-test.

NIAID Data Ecosystem10

NAD + Is a Food Component That Promotes Exit from Dauer Diapause in Caenorhabditis elegans

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分子信号传导

The free-living soil nematode Caenorhabditis elegans adapts its development to the availability of food. When food is scarce and population density is high, worms enter a developmentally arrested non-

NIAID Data Ecosystem20

Identification of Transcription Factor N2::POL II Binding Regions in Late Embryo

转录因子结合位点

线虫发育调控

modENCODE_submission_2436 This submission comes from a modENCODE project of Michael Snyder. For full list of modENCODE projects, see http://www.genome.gov/26524648 Project Goal: We are identifying the

NIAID Data Ecosystem20

Diet-induced developmental acceleration independent of TOR and insulin in C. elegans.

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