Gene expression profiles of tumor-associated macrophages (TAMs) overexpressing miR-511-3p

ChEBI2012-04-01 更新2026-07-21 收录

肿瘤免疫微环境

巨噬细胞调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-34903 数据链接链接失效反馈

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资源简介：

Gene expression profiles of tumor-associated macrophages (TAMs) overexpressing miR-511-3p

创建时间：

2012-04-01

DataSheet_1_Cis- and Trans-Acting Expression Quantitative Trait Loci of Long Non-Coding RNA in 2,549 Cancers With Potential Clinical and Therapeutic Implications.pdf

癌症非编码RNAeQTL分析

肿瘤免疫微环境

Many cancer risk loci act as expression quantitative trait loci (eQTLs) of transcripts including non-coding RNA. Long non-coding RNAs (lncRNAs) are implicated in various human cancers. However, the pa

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Gastric cancer (GC) is one of the most common malignant gastrointestinal tumors worldwide. Pyroptosis was widely reported to exert a crucial function in tumor development. In addition, pyroptosis was

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DataSheet_1_Genomic landscape and expression profile of consensus molecular subtype four of colorectal cancer.zip

结直肠癌分子亚型

肿瘤免疫微环境

BackgroundCompared to other subtypes, the CMS4 subtype is associated with lacking of effective treatments and poorer survival rates. MethodsA total of 24 patients with CRC were included in this study.

NIAID Data Ecosystem00

Table_1_Tumor-Infiltrating Lymphocyte-Based Risk Score for Predicting Prognosis in Gastric Cancer.XLSX

癌症预后预测

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Tumor-infiltrating lymphocytes (TILs) in gastric cancer are closely related to clinical prognosis; however, little is known regarding the immune microenvironment in this disease. Thus, RNA-sequencing

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Table2_Bulk- and single cell-RNA sequencing reveal KIF20A as a key driver of hepatocellular carcinoma progression and immune evasion.xlsx

肝癌基因组

肿瘤免疫微环境

IntroductionKinesin family member 20A (KIF20A) is essential for cell proliferation and is implicated in promoting tumor progression, but its role in hepatocellular carcinoma (HCC) remains poorly studi

NIAID Data Ecosystem00

Gene expression profiles of tumor-associated macrophages (TAMs) overexpressing miR-511-3p

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