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Early Dysregulation of Cell Adhesion and Extracellular Matrix Pathways in Breast Cancer Progression

ChEBI2009-09-08 更新2026-07-21 收录

乳腺癌分子机制

细胞外基质

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-16873 数据链接链接失效反馈

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资源简介：

Early Dysregulation of Cell Adhesion and Extracellular Matrix Pathways in Breast Cancer Progression

应用场景：

创建时间：

2009-09-08

乳腺癌分子机制

RNA调控网络

BRCA-related miRNA sponge interaction network. The network includes 37076 miRNA sponge interactions. (XLSX 484Â kb)

NIAID Data Ecosystem70

matrisome expression matrix

细胞外基质

组织不溶性matrisome分析

Our current study will provide a low-cost, simple, reliable and effective pipeline for tissue insoluble matrisome analysis in the quest to comprehend ECM discovery proteomic studies.

Figshare2023-04-29 更新60

Gene expression analyses of PR action in the mammary gland of ovariectomized mice

乳腺癌分子机制

孕酮信号通路

Beyond demonstrating a critical role for progesterone receptor signaling in normal mammary epithelial proliferation, the progesterone receptor knockout mouse disclosed the progesterone receptor along

NIAID Data Ecosystem30

Shc proteins in DMBA induced breast cancer

乳腺癌分子机制

基因编辑模型

The principal factors that control mammary tumor formation remain insufficiently defined. Shc adaptor proteins play an important role in signaling by three well-established biological markers for prog

NIAID Data Ecosystem70

Dietary ligands diindolylmethane and resveratrol result in diverse ERα signaling not seen after E2 and a subset of diindolylmethane mediated signaling needs concurrent AHR activation.. Dietary ligands diindolylmethane and resveratrol result in diverse ERα signaling not seen after E2 and a subset of diindolylmethane mediated signaling needs concurrent AHR activation.

乳腺癌分子机制

受体信号通路

Inhibitory crosstalk between estrogen receptor alpha (ER alpha ) and aryl hydrocarbon receptor (AHR) regulates 17-estradiol (E2)-dependent breast cancer cell signaling. ER alpha and AHR are transcript

NIAID Data Ecosystem40

Early Dysregulation of Cell Adhesion and Extracellular Matrix Pathways in Breast Cancer Progression

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