Basal-to-inflammatory transition enhances basal cell carcinoma (BCC) therapy resistance via crosstalk with a pro-inflammatory stromal niche [ATAC-Seq]
收藏NIAID Data Ecosystem2026-05-02 收录
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https://www.ncbi.nlm.nih.gov/sra/SRP473097
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The goal of these ATAC-seq experiments was to determine changes in chromatin accesiblity caused by Il1a and Osm ligand treatment of mouse BCC cells at 48h timepoint versus PBS control Overall design: There were two experimental conditions in this ATACseq experiment: (1) PBS control and (2) ligand-treated mouse BCC cells. All samples were performed in biological replicates.
创建时间：
2024-10-10
