Thrombospondin-1: A Pro-Atherosclerotic Protein Augmented by Hyperglycemia

ChEBI2009-08-11 更新2026-07-21 收录

动脉粥样硬化机制

糖尿病心血管并发症

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-17556 数据链接链接失效反馈

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Thrombospondin-1: A Pro-Atherosclerotic Protein Augmented by Hyperglycemia

创建时间：

2009-08-11

Expression data from mouse aorta during and following transient hyperglycaemia. Mus musculus

糖尿病心血管并发症

主动脉基因表达谱

Immediate and early effects of transient hyperglycaemia were examined using fully- reversible transgenic diabetic mice. Transient hyperglycaemia altered expression of 769 arterial genes, of which 200

NIAID Data Ecosystem90

Activation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective. doi: 10.1038/nm.4109

动脉粥样硬化机制

平滑肌细胞转录组

The multiple claims about reactivation of the embryonic stem cell (ESC) pluripotency factor OCT4 in somatic cells are highly controversial due to the fact that there is no direct evidence that OCT4 ha

NIAID Data Ecosystem70

LncRNA ICR regulates endothelium inflammation through feedback activation of NF-κB signaling

动脉粥样硬化机制

血管炎症调控

The responsive and abundant lncRNAs to pro-inflammatory factors in human ECs were screened and validated by RNA sequencing and qPCR. ICAM1-related non-coding RNA (ICR) was identified as the most poten

NIAID Data Ecosystem40

The influence of nt-LNA on cell composition and phenotypees in the atherosclerotic lesion.

动脉粥样硬化机制

单细胞RNA测序

Raw fastq files from paired-end, Illumina-based sequencing on NovaSeq6000 Apoe-/- mice were fed a western diet for 4 weeks and administered saline or nt-LNA (30 mg/kg) weekly. After 4 weeks, aortic a

NIAID Data Ecosystem80

Nicotinic Acid Receptor GPR109A Is Down-Regulated in Human Macrophage-Derived Foam Cells

动脉粥样硬化机制

泡沫细胞

Nicotinic acid (NA) regresses atherosclerosis in human imaging studies and reduces atherosclerosis in mice, mediated by myeloid cells, independent of lipoproteins. Since GPR109A is expressed by human

NIAID Data Ecosystem40

Thrombospondin-1: A Pro-Atherosclerotic Protein Augmented by Hyperglycemia

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