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Neonat cardiomyocytes_hypertrophy_HDAC4
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创建时间：
2011-01-01
相关数据集
Gene expression data from cultured cortical neurons.
We used Affymetrix DNA arrays to investigate the extent to which nuclear HDAC4 accumulation affects neuronal gene expression. Cultured neurons were infected with lentiviruses expressing either wildtyp
NIAID Data Ecosystem80
The β2-subunit of voltage-gated calcium channels inhibits cardiomyocyte hypertrophy through a channel-independent mechanism.
L-type voltage-gated calcium channels (LTCCs) regulate crucial physiological processes in the heart. They are composed of the Cav1 pore-forming subunit and the accessory subunits Cav, Cav2 and Cav. Ca
NIAID Data Ecosystem50
Gene regulation in gastrocnemius muscle of denervated mouse with HDAC4 inducible knockout or class IIa HDAC inhibitior NVS-HD1 treatment
Our class IIa HDAC inhibitor, NVS-HD1, inhibited HDAC4 with less than 1 nM potency while exhibiting >200 fold selectivity on class IIa HDACs compared to class I (HDAC1, 3, 8) and class IIb (HDAC6) HDA
NIAID Data Ecosystem30
Data on sodium tetraborate as a modulus of hypertrophic intracellular signals
Sodium tetraborate is protects cardiomyocytes against hypetrophic stimuli
Mendeley Data2020-07-06 更新60
Gene expression profiling of human-derived primary cardiomyocytes with lncRNA ZNF593-AS 5‘ end overexpression under phenylephrine stimulation. Gene expression profiling of human-derived primary cardiomyocytes with lncRNA ZNF593-AS 5‘ end overexpression under phenylephrine stimulation
The aim of this study is to investigate molecular mechanisms underlying the function of lncRNA ZNF593-AS in cardiomyocyte under phenylephrine stimulation in vitro. Overall design: Human-derived primar
NIAID Data Ecosystem40
