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Characteristics of patients with >10% increase in 53BP1.

NIAID Data Ecosystem2026-03-10 收录

肿瘤生物标志物分析

DNA损伤修复

数据链接：

https://figshare.com/articles/dataset/Characteristics_of_patients_with_10_increase_in_53BP1_/5767887 数据链接链接失效反馈

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资源简介：

Characteristics of patients with >10% increase in 53BP1.

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创建时间：

2018-01-09

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Identifying patients at risk of poor response to neoadjuvant chemoradiotherapy (nCRT) is an emerging clinical need in locally advanced rectal cancer (LARC). SMAD3 is a key player in the chemoradio-res

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53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR)

DNA损伤修复

断裂诱导复制(BIR)

Source data for ‘53BP1 deficiency leads to hyperrecombination using break-induced replication (BIR)’

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Overall survival by NSCLC histology, predictive biomarker testing, and mutation status (EGFR mutation and/or ALK rearrangement).

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Overall survival by NSCLC histology, predictive biomarker testing, and mutation status (EGFR mutation and/or ALK rearrangement).

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SOS mutator activity

DNA损伤修复

突变积累分析

The effect of mismatch repair on the mutational footprint of the SOS mutator activity, by mutation accumulation and whole genome sequencing

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Active DNA demethylation promotes cell fate specification and the DNA damage response [RNA-Seq]

表观遗传调控

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Neurons harbor high levels of endogenous single strand DNA breaks (SSBs) that are targeted to neuronal enhancers and correlate with marks of DNA demethylation. To determine the source of SSBs at neuro

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Characteristics of patients with >10% increase in 53BP1.

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