Autoinflammation with enhanced type I interferon signaling due to DNASE2 mutations
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https://www.ncbi.nlm.nih.gov/sra/ERP022906
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资源简介：
In humans, microbial nucleic acid recognition serves as the major stimulus to an antiviral response, implying a requirement for cellular mechanisms to limit misrepresentation of self nucleic acids as non-self and the subsequent induction of type I interferon-mediated autoinflammation. This RNA-seq study was done on individuals with upregulated type I interferon signaling autoinflammatory disease.
创建时间：
2023-10-13
