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E2F1 loss induces spontaneous tumour development in Rb-deficient epidermis

ChEBI2012-08-20 更新2026-07-21 收录

癌症分子机制

基因功能缺失与肿瘤发生

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-38048 数据链接链接失效反馈

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资源简介：

E2F1 loss induces spontaneous tumour development in Rb-deficient epidermis

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创建时间：

2012-08-20

癌症分子机制

依赖性基因调控

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DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

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WTX overexpression effect on gastric cell line

癌症分子机制

肿瘤抑制基因

We used microarrays to detail the global gene expression in AGS gastric cancer cells overexpressing WTX, a tumor suppressor frequently inactivated in Wilms tumor. Results provide insight into roles fo

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RNA-seq BHT-101 cells and BHT-101 ASH1L KO cell lines

癌症分子机制

转录组学分析

The goal of this study is to assess the role of ASH1 like histone lysine methyltransferase (ASH1L) in the biology of anaplastic thyroid cancer. CRISPR-Cas9 was used to create 4 independent cell lines

NIAID Data Ecosystem30

E2F1 loss induces spontaneous tumour development in Rb-deficient epidermis

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