Multiple tumours from the same patient were analysed for copy number alterations to assess tumour clonality. Seventy-four tumours corresponding to 37 patients were stratified into four groups based on
Introduction Epigenetic modifications such as aberrant DNA methylation has long been associated with tumorogenesis. Little is known, however, about how these modifications appear in cancer progression
In this study, we compared copy number aberration profiles of primary prostate cancerlesions with matching pelvic lymph node metastases of 30 patients to establish clonality between a lymph node metas
