gBRCA1/2 mutations increase the incidence of breast cancer (BC) by interrupting the homologous recombination repair (HRR) pathway. Although gBRCA1 and gBRCA2 BC have similar clinical profiles, differe
DNA-seq was performed on brain and liver tissues taken from wild-type and Polg mutant mice of 6 weeks old, in order to find out the mitochondrial mutation difference and figure out the special feature
Cancer is a genetic disease caused by an accumulation of mutations, however many of these mutations have been identified in pathologically normal tissue. We aim to use laser-capture microscopy (LCM) t