Transcription profiling of human Parkinsons medial and substania nigra
收藏官方服务:
资源简介:
Transcription profiling of human Parkinsons medial and substania nigra
应用场景:
创建时间:
2008-06-16
相关数据集
Examination of gene expression in cerebellum and hippocampus for mouse C57BL/6J WT and nmf205-/-
Ribosome stalling during translation has recently been shown to cause neurodegeneration, yet the signaling pathways triggered by stalled elongation complexes are unknown. To investigate these pathw
NIAID Data Ecosystem100
AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons. AAGGG repeat expansions trigger RFC1-independent synaptic dysregulation in human CANVAS Neurons
This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series
NIAID Data Ecosystem40
Loss of TREM2 reduces hyperactivation of progranulin deficient microglia but not lysosomal pathology [14 months]
GRN haploinsufficiency causes frontotemporal lobar degeneration and results in microglial hyperactivation, lysosomal dysfunction and TDP-43 deposition. To understand the contribution of microglial hyp
NIAID Data Ecosystem30
Characterization of Inducible Models of Tay-Sachs and Related Disease
Tay-Sachs and Sandhoff diseases are lethal inborn errors of acid β-N-acetylhexosaminidase activity, characterized by lysosomal storage of GM2 ganglioside and related glycoconjugates in the nervous sys
NIAID Data Ecosystem40
Next Generation Sequencing facilitated analysis of gene expression differences in the subthalamic nucleus (STN) between Parkinson's and Normal post-mortem human brains
Parkinson’s disease (PD) is a progressive neurodegenerative movement disorder with the characteristic feature of extensive death of dopaminergic neurons in the substantia nigra. The global decrease in
NIAID Data Ecosystem40



