首页/数据集/A non-canonical repressor function of JUN restrains YAP activity and suppresses YAP-dependent liver cancer growth. A non-canonical repressor function of JUN restrains YAP activity and suppresses YAP-dependent liver cancer growth

A non-canonical repressor function of JUN restrains YAP activity and suppresses YAP-dependent liver cancer growth. A non-canonical repressor function of JUN restrains YAP activity and suppresses YAP-dependent liver cancer growth

NIAID Data Ecosystem2026-05-01 收录

肝癌分子机制

Yap信号通路

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJNA988093 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Overall design: Refer to individual Series

应用场景：

创建时间：

2023-06-27

差异基因表达分析

Yap信号通路

Previously, lncRNA Malat1 knockout mice were generated by insertional inactivation. By crossing this line to MMTV-PyMT mammary tumor mouse model, we produced PyMT;Malat1 wild-type (WT) and PyMT;Malat1

NIAID Data Ecosystem40

Genomic profiles of mouse liver tissue harboring deletion of RB and/or p53, untreated or post-hepatocarcinogen treatment [expression data]

肝癌分子机制

肿瘤抑制基因

The retinoblastoma (RB) and p53 tumor suppressors are critical regulators of the cell cycle with profound impact on both normal cell biology and disease etiology. In the context of human cancers, comp

NIAID Data Ecosystem30

Mir-184 Post-Transcriptionally Regulates SOX7 Expression and Promotes Cell Proliferation in Human Hepatocellular Carcinoma

肝癌分子机制

miRNA功能研究

Hepatocellular carcinoma (HCC) is one of the most common human malignancies and the third leading cause of cancer mortality worldwide. The development and progression of HCC is a complicated process,

Figshare2016-01-18 更新20

Transcriptional effects of YAP/TAZ knockdown in fibroblasts. Transcriptional effects of YAP/TAZ knockdown in fibroblasts

成纤维细胞衰老

Yap信号通路

Gene expression profiles of the cells composing the dorsal skin of young control and YAP/TAZ knockout mice with the skin of a old mouse. Overall design: Single cell profiles were derived from the dors

NIAID Data Ecosystem40

CRTC2 Forms Co-condensates with YTHDF2 that Enhance Translational Efficiency of m6A-Modified mRNAs to Drive Hepatocarcinogenesis and Lenvatinib Resistance (RNA-seq)

肝癌分子机制

mRNA翻译调控

As the third most common cause of cancer-related mortality, hepatocellular carcinoma (HCC) is a global health concern. Despite its prevalence, treatment options are limited, underscoring the need to i

NIAID Data Ecosystem20

A non-canonical repressor function of JUN restrains YAP activity and suppresses YAP-dependent liver cancer growth. A non-canonical repressor function of JUN restrains YAP activity and suppresses YAP-dependent liver cancer growth

资源简介：

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