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M1-like BV2 cells treated with siNC-hNVs or siMETTL3-hNVs
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创建时间：
2026-01-13
相关数据集
Genetic Rescue of X-Linked Retinoschisis Mouse (Rs1-/y) Retina Induces Quiescence of the Retinal Microglial Inflammatory State Following AAV8- RS1 Gene Transfer and Identifies Gene Networks Underlying Retinal Recovery
To understand RS1 gene interaction networks in the X-linked retinoschisis (XLRS) mouse retina (Rs1-/y), we analyzed the transcriptome by RNA sequencing before and after in vivo expression of exogenous
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Age and dietary fiber effect on mouse brain cells. Age and dietary fiber effect on mouse brain cells
With increasing age microglia shift toward a pro-inflammatory phenotype and become hyperresponsive to inflammatory stimuli, disrupting brain homeostasis. Soluble fibers have been suggested as a dietar
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m6A-seq profiling in Mettl3 Ctrl and dTAG-13 treated mESCs
dTAG-13 treated METTL3_FKBP12(F36V) is sufficient to functionally deplete m6A methylation catalysed by METTL3/14 complex. We would like to confirm the functional m6A loss by dTAG-13 treatment in the M
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High depth sequencing of BrU-labeled nascent RNA in METTL3 depleted cells.. High depth sequencing of BrU-labeled nascent RNA in METTL3 depleted cells.
BrU incorporation into nascent RNA followed by immunoprecipitation with a BrU specific antibody. The nascent RNA was subsequently eluted with BrU competition. Overall design: HEK293T cells, treated wi
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DataSheet_1_Extracellular Nucleosomes Accelerate Microglial Inflammation via C-Type Lectin Receptor 2D and Toll-Like Receptor 9 in mPFC of Mice With Chronic Stress.docx
Damage-associated molecular patterns (DAMPs) are the primary promoter of progressive neuroinflammation and are associated with chronic stress-related emotional disorders. The present study investigate
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