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Deacetylation of HP1g enhances multiple myeloma drug resistance through DNA damage repair and liquid-liquid phase separation

NIAID Data Ecosystem2026-05-01 收录

多发性骨髓瘤耐药机制

表观遗传调控

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE176547 数据链接链接失效反馈

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资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

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创建时间：

2024-01-27

表观遗传调控

甲基化反应机制

DNA methylation at the 5-position of cytosine (5mC) is a crucial epigenetic mark in regulating biological processes including gene silencing, gene imprinting, and X chromosome inactivation. It has lon

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Epigenetic gene regulation during astrocyte development

胶质细胞发育

表观遗传调控

Numerous literatures report similar time points are important in astrocyte development and transition from neurogenesis to gliogenesis. However, these developmental time points have not been studied i

NIAID Data Ecosystem00

H3 Acetylation and H2A.Z Levels Interplay to Regulate Gene Expression and Plant Development

表观遗传调控

发育分子机制

H2A.Z is a conserved histone variant that plays essential roles in various DNA-templated processes across eukaryotes. H2A.Z and histone post-translational modifications (PTMs) exert a bidirectional an

NIAID Data Ecosystem00

The mitochondrial protease LonP1 promotes proteasome inhibitor resistance in multiple myeloma.

多发性骨髓瘤耐药机制

蛋白酶体抑制剂转录组分析

Purpose: We report the NGS-derived transcriptome profiling (paired-end RNA-seq) following proteasome inhibition in the multiple myeloma cell line MM.1S. Methods: MM.1S cells were treated for six hours

NIAID Data Ecosystem00

A TAD boundary is preserved upon deletion of the CTCF-rich Firre locus [RNA-Seq, HiC_C2C12, ChIP-Seq]

基因组三维结构

表观遗传调控

The binding of the transcriptional regulator CTCF to the genome has been implicated in the formation of topologically associated domains (TADs). However, the general mechanisms of folding the genome i

NIAID Data Ecosystem00

Deacetylation of HP1g enhances multiple myeloma drug resistance through DNA damage repair and liquid-liquid phase separation

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