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ADAM9 Up-regulates N-Cadherin via MiR-218 Suppression in Lung Adenocarcinoma Cells

ChEBI2016-07-03 更新2026-07-12 收录

肺腺癌分子机制

肿瘤转移调控

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-52051 数据链接链接失效反馈

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资源简介：

ADAM9 Up-regulates N-Cadherin via MiR-218 Suppression in Lung Adenocarcinoma Cells

应用场景：

创建时间：

2016-07-03

肺腺癌分子机制

HIF-1α信号通路调控

G protein-coupled receptor kinase 6 (GRK6) is expressed in various tissues and is involved in the development of several diseases including lung cancer. We previously reported that GRK6 is down-regula

NIAID Data Ecosystem120

Additional file 15 of DUSP5 regulated by YTHDF1-mediated m6A modification promotes epithelial-mesenchymal transition and EGFR-TKI resistance via the TGF-β/Smad signaling pathway in lung adenocarcinoma

癌症信号通路

肺腺癌分子机制

Supplementary Material 15

Figshare2024-08-18 更新60

A micropeptide encoded by DSP-AS1 promotes lung adenocarcinoma by facilitating YTHDF1-induced oncogene translation initiation

肺腺癌分子机制

肽功能研究

A micropeptide encoded by DSP-AS1 promotes lung adenocarcinoma by facilitating YTHDF1-induced oncogene translation initiation

integrated proteome resources50

Investigation on molecular mechanism underlying the dynamic evolution from preneoplasia to invasive lung adenocarcinoma

肺腺癌分子机制

肿瘤微环境动态

The cell ecology and spatial niche implicated in the dynamic and sequential process of lung adenocarcinoma (LUAD) from adenocarcinoma in situ (AIS) to minimally invasive adenocarcinoma (MIA) and subse

NIAID Data Ecosystem90

Knockdown of FOXP1 promotes the development of lung adenocarcinoma. human

肺腺癌分子机制

趋化因子信号通路

Lung cancer is one of the most common cancers in the world, which accounts for about 27% of all cancer deaths. However, the mechanisms underlying the pathogenesis of lung cancer cells remain largely e

NIAID Data Ecosystem60

ADAM9 Up-regulates N-Cadherin via MiR-218 Suppression in Lung Adenocarcinoma Cells

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