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Transposon mutagenesis reveals fludarabine-resistance mechanisms in chronic lymphocytic leukemia

ChEBI2016-03-31 更新2026-07-12 收录

白血病耐药机制

转座子诱变

数据链接：

https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-GEOD-75844 数据链接链接失效反馈

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资源简介：

Transposon mutagenesis reveals fludarabine-resistance mechanisms in chronic lymphocytic leukemia

应用场景：

创建时间：

2016-03-31

白血病耐药机制

RNAm6A修饰分析

N6-Methyladenosine (m6A) is the most abundant post-transcriptional modification in eukaryotes, the imbalance of which is reported to be associated with various pathological processes, including drug r

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Crystal structure of the double mutant (D52N/L375F) of the full-length NT5C2 in the active state

白血病耐药机制

蛋白质结构解析

Crystal structure of the double mutant (D52N/L375F) of the full-length NT5C2 in the active state Descriptor: 1,2-ETHANEDIOL, ADENOSINE-5'-TRIPHOSPHATE, Cytosolic purine 5'-nucleotidase, ... Authors: F

Protein Data Bank Japan2023-10-11 更新50

Data_Sheet_1_Acute Myeloid Leukemia Chemo-Resistance Is Mediated by E-selectin Receptor CD162 in Bone Marrow Niches.PDF

白血病耐药机制

肿瘤微环境靶向治疗

The interactions of leukemia cells with the bone marrow (BM) microenvironment is critical for disease progression and resistance to treatment. We have recently found that the vascular adhesion molecul

NIAID Data Ecosystem40

Crystal structure of the double mutant (R39Q/D52N) of the full-length NT5C2 in the active state

蛋白质晶体

白血病耐药机制

Crystal structure of the double mutant (R39Q/D52N) of the full-length NT5C2 in the active state Descriptor: ADENOSINE-5'-TRIPHOSPHATE, Cytosolic purine 5'-nucleotidase, MAGNESIUM ION, ... Authors: For

Protein Data Bank Japan2023-10-11 更新60

Gene trapping

蚊子基因捕获

转座子诱变

RNA from 200 pooled individually-extracted An. stephensi that demonstrated a gene trap remobilizable event.

NIAID Data Ecosystem50