Pan-genomic analysis of hypoxic HIF-1b binding by ChIP-seq in cancer cell lines Overall design: ChIP_seq analysi of HIF-1b binding in 4 cancer cell lines incubated in 0.5% hypoxia for 16 hours

NIAID Data Ecosystem80

Jak2V617F Reversible Activation Shows an Essential Requirement for Jak2V617F in Myeloproliferative Neoplasms. Jak2V617F Reversible Activation Shows an Essential Requirement for Jak2V617F in Myeloproliferative Neoplasms

骨髓增殖性肿瘤

JAK2V617F突变

Janus kinases (JAKs) mediate cytokine signaling, cell growth and hematopoietic differentiation. Gain-of-function mutations activating JAK2 signaling are seen in the majority of myeloproliferative neop

NIAID Data Ecosystem60

ChIP-Seq of Transcriptional Components in Ly1 DLBCL and Colon carcinoma. Homo sapiens

癌症表观基因组学

转录调控ChIP-seq

Discovery of the genome-wide location of proteins using ChIP-Seq has allowed global mapping of the key transcription factors and chromatin regulators that control gene expression programs in various c

NIAID Data Ecosystem90

Genome-wide profiling of the functional DNA methylation landscape at base-pair resolution in human cancer types [BS-seq]

癌症表观基因组学

差异甲基化分析

Whole genome bisulphite sequencing of 13 human cancer samples and 9 normal controls. The main goal is to find the Diffrenetial methylated regions (DMR) at Genome wide level in different tissues and ca

NIAID Data Ecosystem80

Evolution of the cancer epigenome in myeloproliferative neoplasms 2

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