KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer.
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2023-09-07
相关数据集
KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer (atac batch3)
Using a CRISPR-based autochthonous SCLC GEMM to study the consequences of KDM6A/UTX inactivation, we found that KDM6A inactivation induced plasticity from ASCL1 to NEUROD1 resulting in SCLC tumors wit
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KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer. KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer
Using a CRISPR-based autochthonous SCLC GEMM to study the consequences of KDM6A/UTX inactivation, we found that KDM6A inactivation induced plasticity from ASCL1 to NEUROD1 resulting in SCLC tumors wit
NIAID Data Ecosystem30
KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer (scatac). KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer (scatac)
Small cell lung cancer (SCLC) exists broadly in four molecular subtypes: ASCL1, NEUROD1,POU2F3, and Inflammatory. Initially SCLC subtypes were thought to be mutually exclusive, butrecent evidence show
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Small Cell Lung Cancer Subtype Plasticity is Regulated by KDM6A/UTX [ChIP-Seq]. Small Cell Lung Cancer Subtype Plasticity is Regulated by KDM6A/UTX [ChIP-Seq]
Using a CRISPR-based autochthonous SCLC GEMM to study the consequences of KDM6A/UTX inactivation, we found that KDM6A inactivation induced plasticity from ASCL1 to NEUROD1 resulting in SCLC tumors wit
NIAID Data Ecosystem30
KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer (atac batch1). KDM6A Epigenetically Regulates Subtype Plasticity in Small Cell Lung Cancer (atac batch1)
Small cell lung cancer (SCLC) exists broadly in four molecular subtypes: ASCL1, NEUROD1,POU2F3, and Inflammatory. Initially SCLC subtypes were thought to be mutually exclusive, butrecent evidence show
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