HNRNPM-regulated splicing dependencies in prostate cancer
收藏NIAID Data Ecosystem2026-03-11 收录
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2019-12-26
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HNRNPM-regulated splicing dependencies in prostate cancer [RNA-Seq]. HNRNPM-regulated splicing dependencies in prostate cancer [RNA-Seq]
Cancer cells are differentially dependent on the splicing machinery compared to normal untransformed cells. The splicing machinery thus presents a potential therapeutic target in cancer. To identify s
NIAID Data Ecosystem40
RNAseq analysis of ESRP regulated splicing events in prostate cancer. RNAseq analysis of ESRP regulated splicing events in prostate cancer
Progression of prostate cancer -the most frequent cancer in men- is driven by androgen steroid hormones, and delayed by androgen deprivation therapy (ADT). Androgens control transcription in prostate
NIAID Data Ecosystem30
Targeting the CLK2/SRSF9 splicing axis in prostate cancer leads to decreased ARV7 expression in an rs5918762 allele-dependent manner (ONT long read sequencing)
The Cdc2-like kinase (CLK)-family is a regulator of the splicing process. Here, we report the effect of Lorecivivint (SM04690), a CLK/DYRK inhibitor, on alternative splicing in the prostate cancer c
NIAID Data Ecosystem20
HNRNPM-regulated splicing dependencies in prostate cancer [RNA-Seq]
Cancer cells are differentially dependent on the splicing machinery compared to normal untransformed cells. The splicing machinery thus presents a potential therapeutic target in cancer. To identify
NIAID Data Ecosystem30
A RNA splicing factor regulating splicing in androgen receptor promotes malignancy in prostate cancer. Homo sapiens strain:22Rv1
RNA splicing creates multiple transcript variants from one pre-mRNA via removing introns or some exons. Although an impediment in Androgen-Androgen receptor (AR) axis effectively represses the growth
NIAID Data Ecosystem20
