Tlr5 deficiency exacerbates lupus-like disease in the MRL/lpr mouse model
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https://www.ncbi.nlm.nih.gov/bioproject/PRJNA1021666
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资源简介：
We hypothesized that bacterial flagellin-mediated activation of TLR5 would trigger SLE-like autoimmunity in lupus-prone MRL/lpr mice. To provide a comprehensive exploration of the intricate relationship between a leaky gut, flagellin-TLR5 signaling, and their potential impact on the development and progression of disease in SLE, we have assessed the development of autoimmune disease in our newly generated Tlr5-deficient lupus-prone MRL/lpr mice.
创建时间：
2023-09-27
