EBF2 links KMT2D-mediated H3K4me1 to suppress pancreatic cancer progression via upregulating KLLN [ChIP-seq]
收藏NIAID Data Ecosystem2026-05-10 收录
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https://www.ncbi.nlm.nih.gov/sra/SRP449505
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资源简介：
ChIP-seq revealed a strong reduction of KMT2D, H3K4me1 and EBF2 signals at the transcription start site (TSS) and promoter regions of genes that were downregulated in KMT2D-depleted (KMT2D-KD) cells Overall design: Chromatin immunoprecipitation DNA-sequencing (ChlP-seq) for KMT2D, EBF2 as well as the histone modifications H3K4me1 in negative control and KMT2D knowndown SW1990 cells.
创建时间：
2026-02-24
