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Oncogene inactivation induced senescence facilitates tumor relapse. Oncogene inactivation induced senescence facilitates tumor relapse

NIAID Data Ecosystem2026-05-02 收录

肿瘤复发机制

癌基因失活诱导衰老

数据链接：

https://www.ncbi.nlm.nih.gov/bioproject/PRJEB78692 数据链接链接失效反馈

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资源简介：

Oncogene inactivation induces senescence and facilitates tumor relapse in vivo

应用场景：

创建时间：

2024-08-05

胶质瘤单细胞图谱

肿瘤复发机制

Most clinical trials for glioblastoma (GBM) enroll patients at recurrence. However, most pre-clinical animal models are of primary disease. Most studies have used tissues from primary tumors. Knowledg

NIAID Data Ecosystem230

Reawakening of Dormant Tumor Cells by Modified Lipids derived from Stress Activated Neutrophils

肿瘤休眠细胞再激活

肿瘤复发机制

Tumor recurrence years after seemingly successful treatment of primary tumors is one of the major causes of mortality in cancer patients. Reactivation of dormant tumor cells is largely responsible for

NIAID Data Ecosystem90

Impact of disseminated neuroblastoma cells on the identification of the relapse seeding clone

神经母细胞瘤克隆演化

肿瘤复发机制

Background: Since the high relapse rate is considered to be responsible for the poor survival of stage M neuroblastoma patients, we tested whether the genomic information of bone marrow-derived dissem

NIAID Data Ecosystem70

High-complexity cellular barcoding identifies clonal substitution as a hallmark of local recurrence in a surgical model of head and neck cancer. HNSCC RTICs Exome Sequencing

肿瘤复发机制

克隆动态

The survival prognosis for head and neck squamous cell carcinoma (HNSCC) remains dismal mainly because of insufficient local and loco-regional control resulting in a significant number of treatment fa

NIAID Data Ecosystem90

Additional file 1 of A longer time to relapse is associated with a larger increase in differences between paired primary and recurrent IDH wild-type glioblastomas at both the transcriptomic and genomic levels

神经胶质瘤异质性分析

肿瘤复发机制

Supplementary Material 1

DataCite Commons2024-08-17 更新80