LKB1 drives stasis and C/EBP-mediated reprogramming to an alveolar type II fate in lung cancer

NIAID Data Ecosystem2026-03-13 收录

癌细胞重编程

肺泡II型细胞分化

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE179560 数据链接链接失效反馈

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This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

创建时间：

2022-03-23

Additional file 1: of Effects of PKM2 on global metabolic changes and prognosis in hepatocellular carcinoma: from gene expression to drug discovery

癌细胞重编程

靶向药物研发

Table S1. Differentially expressed genes in high-PKM2 group. Table S2. Differentially expressed genes in low-PKM2 group. Table S3. Descriptive statistics of PKM2 and other clinical features grouped by

NIAID Data Ecosystem70

Long non-coding RNAs are involved in the activation of glycolysis in colorectal cancer

癌细胞重编程

非编码RNA功能

Previous studies have documented that long non-coding RNAs participate in a wide-spectrum of biological processes. We hypothesized that long non-coding RNAs may promote glycolysis and tumorigenesis in

NIAID Data Ecosystem20

Activation of phosphatidylcholine-cycle enzymes in human epithelial ovarian cancer cells

癌细胞重编程

卵巢癌分子靶点

Altered phosphatidylcholine (PC) metabolism in epithelial ovarian cancer (EOC) can provide choline-based imaging approaches as powerful tools to improve diagnosis and identify new therapeutic targets.

NIAID Data Ecosystem20

Y.T.,SUCLA2-coupled regulation of GLS succinylation and activity counteracts oxidative stress of tumor cells

癌细胞重编程

癌症分子机制

Glutaminase regulates glutaminolysis for cancer cell proliferation. However, the mechanism underlying glutaminase activity regulation is largely unknown. Here we demonstrated that the kidney-type glut

NIAID Data Ecosystem20

The Long Non Coding RNA SAMMSON is a regulator of chemosensitivity and metabolic orientation in doxorubicin resistant breast cancer cells

癌细胞重编程

化疗耐药机制

Despite improvements in therapeutic strategies for treating breast cancers, tumor relapse and chemoresistance remain major issues in patient outcomes. Indeed, cancer cells display a metabolic plastici

NIAID Data Ecosystem20

LKB1 drives stasis and C/EBP-mediated reprogramming to an alveolar type II fate in lung cancer

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