Rhinoviruses and enterovirus-D68 replicate more robustly in respiratory tissues originating from asthmatic patients compared to non-asthmatic controls

Investigation of the mechanism leading to differential rhinovirus infection in airway epithelia from healty versus asthmatic patients Overall design: Transcriptomic comparison of Human airway epithelia from healthy versus asthmatic patients infected with Rhinoviruses RV-48 or RV-15. Non-infected tissues from the same groups (mock) have been used as negative controls