High Yap and Mll1 promote a persistent regenerative cell state induced by Notch signaling and loss of p53

ChEBI2021-06-04 更新2026-07-12 收录

细胞周期调控

Notch信号通路

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https://www.ebi.ac.uk/biostudies/ArrayExpress/studies/E-MTAB-6588 数据链接链接失效反馈

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High Yap and Mll1 promote a persistent regenerative cell state induced by Notch signaling and loss of p53

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2021-06-04

In-phase oscillation of global regulons is orchestrated by a pole-specific organizer. Caulobacter vibrioides NA1000

细胞周期调控

极性发育分子机制

Cell fate determination in the asymmetric bacterium Caulobacter crescentus (Caulobacter) is triggered by the localization of the developmental regulator SpmX to the old (stalked) cell pole during the

NIAID Data Ecosystem70

A kinase-phosphatase switch transduces environmental information into a bacterial cell cycle circuit. Caulobacter vibrioides

细胞周期调控

环境胁迫信号转导

The bacterial cell cycle has been extensively studied under standard growth conditions. How it is modulated in response to environmental changes remains poorly understood. Here, we demonstrate that th

NIAID Data Ecosystem60

DataSheet4_PRR11 promotes cell proliferation by regulating PTTG1 through interacting with E2F1 transcription factor in pan-cancer.zip

癌症分子机制

细胞周期调控

The upregulated proline rich 11 (PRR11) plays a critical role in cancer progression. The relevant biological functions of PRR11 in pan-cancer development are not well understood. In the current study,

NIAID Data Ecosystem30

Additional file 16 of STAU2 protein level is controlled by caspases and the CHK1 pathway and regulates cell cycle progression in the non-transformed hTERT-RPE1 cells

细胞周期调控

蛋白相互作用分析

Additional file 16: Table S9. Gene ontology (GO) of STAU2 interactors that show reduced proximity to STAU2 in hTERT-RPE1 cells treated with the CHK1 inhibitor PF47 compared to untreated cells. List of

Figshare2021-03-05 更新60

Arrest of G(1)-S Progression by the p53-Inducible Gene PC3 Is Rb Dependent and Relies on the Inhibition of Cyclin D1 Transcription

细胞周期调控

肿瘤抑制基因

The p53-inducible gene PC3 (TIS21, BTG2) is endowed with antiproliferative activity. Here we report that expression of PC3 in cycling cells induced accumulation of hypophosphorylated, growth-inhibitor

PubMed Central30

High Yap and Mll1 promote a persistent regenerative cell state induced by Notch signaling and loss of p53

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