首页/数据集/Transcriptional regulation loss disturbs the brain function and indicates detrimental clinical outcomes of Alzheimer's disease

Transcriptional regulation loss disturbs the brain function and indicates detrimental clinical outcomes of Alzheimer's disease

NIAID Data Ecosystem2026-03-14 收录

阿尔茨海默病转录研究

神经退行性分子机制

数据链接：

https://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE129041 数据链接链接失效反馈

官方服务：

资源简介：

This SuperSeries is composed of the SubSeries listed below. Refer to individual Series

应用场景：

创建时间：

2023-02-07

神经退行性分子机制

脑源因子调控

Patterns of gene expression in the aged entorhinal cortex and hippocampus were examined one month after entorhinal administration of BDNF lentivirus. Whole-genome patterns of expression were examined

NIAID Data Ecosystem30

CryoEM structure of amplified alpha-synuclein fibril class B type I with extended core from DLB case VII

α-突触核蛋白纤维研究

神经退行性分子机制

CryoEM structure of amplified alpha-synuclein fibril class B type I with extended core from DLB case VII Descriptor: Alpha-synuclein Authors: Zhou, Y, Sokratian, A, Xu, E, Viverette, E, Dillard, L, Yu

Protein Data Bank Japan2024-06-12 更新10

Comprehensive Analysis of Gene Expression Patterns in Friedreich's Ataxia Fibroblasts by RNA Sequencing Reveals Altered Levels of Protein Synthesis Factors and Solute Carriers

神经退行性分子机制

转录组分析

Friedreich's ataxia (FRDA) is an autosomal recessive neurodegenerative disease usually caused by large homozygous expansions of GAA repeat sequences in intron 1 of the frataxin (FXN) gene. FRDA patie

NIAID Data Ecosystem10

Global gene profiling of (-)-Epigallocatechin-3-gallate (EGCG)-induced neuronal death. Mus musculus

神经退行性分子机制

药物神经保护

Alzheimer’s disease (AD) is the most common form of adult-onset dementia with severe intellectual deterioration and is characterised by the accumulation of the amyloid-β (Aβ) peptides and the presence

NIAID Data Ecosystem30

Expanded gene expression changes in inducible TDP-43 flies.

神经退行性分子机制

TDP-43蛋白相关基因表达

Genes are from same experiment and analysis as in Table 3, with less stringent criteria to identify potentially important genes. nACR, nicotinic acetycholine receptor.

Figshare2015-12-02 更新10

Transcriptional regulation loss disturbs the brain function and indicates detrimental clinical outcomes of Alzheimer's disease

资源简介：

相关数据集